Welcoem to posts!!
in the future - u will be able to do some more stuff here,,,!! like pat catgirl- i mean um yeah... for now u can only see others's posts :c
Stay Tuned. 2025-2030 is going to be groundbreaking years for pathology360 !! Something B-I-G is coming in 2025!!
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PT normal, APTT prolonged—what’s the secret behind this classic lab finding in Hemophilia? Explained in the uploaded video of the day.
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#CaseOfTheDay : A 10-year-old boy with severe Hemophilia A presents with recurrent swelling and pain in his right knee. His mother reports frequent episodes of joint stiffness, particularly in the morning. On examination, the knee is swollen with reduced range of motion and mild deformity. Radiographs reveal joint space narrowing and subchondral bone changes. MRI shows synovial hypertrophy.
🚨Question : What’s the MOST likely way to prevent this disorder?? Answer : Prophylactic clotting factor replacement. More details about haemophiliac arthropathy have been shared in the video posted earlier.
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Why🤔 does Cobweb Coagulum form in CSF samples in TB meningitis but not in bacterial or viral meningitis?
• 🔺Concept: It’s due to a high concentration of protein in the CSF in TB meningitis. This elevated protein level is significantly higher than in viral or bacterial meningitis.
• In TB meningitis, the CSF often has protein levels above 500 mg/dL.
• Fibrin, a protein in the CSF, forms a mesh-like structure resembling a spider’s web – hence, the term “Cobweb Coagulum.”
Key Point: Cobweb Coagulum is NOT diagnostic of TB meningitis alone.
• Definitive diagnosis requires NAAT (Nucleic Acid Amplification Test), AFB smear, and culture.
• Clinical Insight: Spotting Cobweb Coagulum can be a visual clue but needs confirmation through advanced testing.
• Pathology Pearl: High protein, cobweb signs? Think 🤔TB meningitis but confirm 😀to be sure! #pathology360riginal :1-years-ago; #pathology360 #DrKishoreBanerjee #TBMeningitis #Medicos #doctor #healthcare #InfectiousDisease; Associate Professor Dr Kishore G Banerjee, MD(AIIMS, Delhi)
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Why is serum amylase elevated in perforated peptic ulcer disease (PUD)? 🧐
Many confuse it with pancreatitis, but here’s the mechanism:
1️⃣ Most peptic ulcers occur in the duodenum, where pancreatic enzymes reach for digestion.
2️⃣ When the ulcer perforates, fluid rich in pancreatic enzymes (like amylase) leaks into the peritoneal cavity.
3️⃣ These enzymes are absorbed through the peritoneal lymphatics, leading to elevated serum amylase levels.
⚠️ Both conditions present with acute abdomen, but serum amylase elevation is 🔺NOT specific to pancreatitis—it’s a key feature of perforated duodenal ulcers too.
🔑 Knowing this avoids 🚨diagnostic confusion!
#Pathology360 #MedicalConcepts #PUD #medicine #ClinicalPearls #MedEd #medicos ; Associate Professor Dr Kishore G Banerjee|| MBBS, MD(AIIMS, New Delhi), member USCAP.
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Uploaded the concept-videos on Stress Ulcers, Curling Ulcer & Cushing ulcer. Peptic Ulcer Vs Stress Ulcers.
🔺Curling Ulcer = Stress ulcer associated with : Severe burns/hypovolemia>> massive loss of plasma volume >> reduced blood-supply to stomach>> reduced blood supply to gastric mucosa >> weakens “ protective” mucosal barrier >> ulcer = Curling Ulcer
🔺Cushing ulcer = A stress ulcer caused by brain injury; brain 🧠 injury/trauma> Excess stimulation of Vagus nerve > excess release of ACh ( Acetylcholine)> excess ACh over-stimulates gastric parietal cells to release EXCESS acid >> damage to stomach mucosa >> ulcer forms = Cushing ulcer ; Explained the details in the video. Associate Professor Dr Kishore G Banerjee|| MBBS, MD(AIIMS, New Delhi)
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Uploaded this latest video in YouTube shorts. The Link Between Rapid Weight Loss and Gallstone Formation: 2-key reasons. #medico #doctor #pathology360 #GallstoneRisk
#RapidWeightLoss
#GallbladderHealth
#MedicalInsights
#GallstonesExplained
#Pathology360
#WeightLossTips
#StoneFormation
#DigestiveHealth
#HealthyWeightLoss
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🔥 Cushing Ulcer Vs Curling Ulcer: The 🔺Stress Ulcer Duo 🔥
1. Curling Ulcer
Severe Burns → Decreased plasma volume → Less blood flow to the stomach → Stomach mucosal barrier weakens → Ulcers form= CURLING ulcer.
2. Cushing Ulcer
Brain injury → Vagus nerve overstimulates → Acetylcholine surge → → Stomach acid overload → Ulcers develop = Cushing ulcer( Key 🔑 concept : Increased vagal stimulation leads to increased production of stomach acid)
Key takeaway: When the body’s stressed, the stomach suffers! 🩺 #pathology360 #StressUlcers #medicine #medico #gastricPathology ; Associate Professor Dr Kishore G Banerjee || MBBS, MD(AIIMS, Delhi)
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Have you ever wondered why some people develop diarrhea after gallbladder removal? Is it just a coincidence, or is there a logic behind it? Let’s uncover the mystery together! Full, detailed video on my YouTube channel ( pathology360)
The gallbladder plays a crucial role in digestion by storing bile, but what happens when it’s removed? For many, life without a gallbladder comes with an unexpected surprise—post-cholecystectomy diarrhea. In this video, we’ll explore why this happens, the science behind bile acid metabolism, and how you can manage it effectively. Stay tuned—it’s simpler than you think!
Associate Professor Dr Kishore G Banerjee, MBBS, MD(AIIMS, Delhi)
#PostCholecystectomyDiarrhea
#GallbladderRemovalFacts
#BileAcidMetabolism
#MedicalEducation
#MedStudentLife
#DoctorsOfInstagram
#PathophysiologyExplained
#SurgicalComplications
#MedSchoolTips
#MedicalReels
#LearnMedicine
#DigestiveHealth
#MedGram
#FutureDoctors #pathology360 #drkishorebanerjee
#MedicalProfessionals
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What’s ✅Gallstone Ileus?? : explained it here. Gallstone ileus occurs as a result of complication of gallstone formation. Due to gallstone >> cholecystitis(= inflammation occurs in Gall bladder due to stone formation) occurs where persistent inflammation erodes the gallbladder wall, leading to the formation of a ✅biliary-enteric fistula, most commonly with the duodenum. Through this fistula, a large gallstone escapes into the gastrointestinal tract. The gallstone travels through the intestines but becomes lodged in a narrow segment, typically the ileum, causing mechanical obstruction and resulting in gallstone ileus. #GallstoneIleus
#BiliaryFistula
#ObstructionAlert
#RiglersTriad
#GallstoneTrouble
#PathologySimplified
#SurgicalEmergency
#IntestinalObstruction
#RareButReal #medicine #medicos #medstudygram
#pathology360riginal ; Associate Professor Dr Kishore G Banerjee|| MBBS, MD(AIIMS, New Delhi)
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Associate Professor Dr Kishore G Banerjee. MBBS, MD in Pathology( AIIMS, New Delhi, India).Member of USCAP ( United States And Canadian Academy of Pathology).All India 🇮🇳 61 Rank (General category)in AIIMS MD/MS Entrance Examination. Choose pathology form specialisation out of passion leaving all other clinical subjects.
Currently,Associate Professor in Pathology Department in the faculty of medicine,Manipal University College Malaysia (MUCM)🇲🇾 . Consultant pathologist.l national and international experience as an educator for medical students.
Loves to simplify the complex concepts of medicine and pathology. I strongly believe that simplicity is the ultimate sophistication. Passionate about sharing knowledge of pathology & medicine. NTSE national level scholar.
Instagram account: pathology360
THIS CHANNEL IS FOR EDUCATIONAL PURPOSES ONLY; not for Consultation.